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Published online
doi:10.1083/jcb.200903134
The Journal of Cell Biology, Vol. 185, No. 2, 189-191
The Rockefeller University Press, 0021-9525 $30.00
© Letai
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Puma strikes Bax



Anthony Letai

Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA 02115

Correspondence to Anthony Letai: anthony_letai{at}dfci.harvard.edu

The commitment to programmed cell death via apoptosis is largely made upon activation of the proapoptotic mitochondrial proteins Bax or Bak. In this issue, Gallenne et al. (Gallenne, C., F. Gautier, L. Oliver, E. Hervouet, B. Noël, J.A. Hickman, O. Geneste, P.-F. Cartron, F.M. Vallette, S. Manon, and P. Juin. 2009. J. Cell Biol. 185:279–290) provide evidence that the p53 up-regulated modulator of apoptosis (Puma) protein can directly activate Bax.

© 2009 Letai
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Related Article

Bax activation by the BH3-only protein Puma promotes cell dependence on antiapoptotic Bcl-2 family members
Tristan Gallenne, Fabien Gautier, Lisa Oliver, Eric Hervouet, Belinda Noël, John A. Hickman, Olivier Geneste, Pierre-François Cartron, François M. Vallette, Stephen Manon, and Philippe Juin
J. Cell Biol. 2009 185: 279-290. [Abstract] [Full Text] [PDF]



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