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Published online
doi:10.1083/jcb.200904033
The Journal of Cell Biology, Vol. 185, No. 4, 573-575
The Rockefeller University Press, 0021-9525 $30.00
© Ball et al.
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Revisiting the role of heterochromatin protein 1 in DNA repair



Alexander R. Ball, Jr. and Kyoko Yokomori

Department of Biological Chemistry, School of Medicine, University of California, Irvine, Irvine, CA 92697

Correspondence to Kyoko Yokomori: kyokomor{at}uci.edu

Heterochromatin protein 1 (HP1) is a conserved factor critical for heterochromatin organization and gene silencing. It is recruited to chromatin by its direct interaction with H3K9me (methylated lysine 9 residue of histone H3), an epigenetic mark for silenced chromatin. Now, Luijsterburg et al. (Luijsterburg, M.S., C. Dinant, H. Lans, J. Stap, E. Wiernasz, S. Lagerwerf, D.O. Warmerdam, M. Lindh, M.C. Brink, J.W. Dobrucki, et al. 2009. J. Cell Biol. 185:577–586) reveal a new H3K9me-independent role for HP1 in the DNA damage response, which is distinct from the one recently reported by Ayoub et al. (Ayoub, N., A.D. Jeyasekharan, J.A. Bernal, and A.R. Venkitaraman. 2008. Nature. 453:682–686).

© 2009 Ball and Yokomori
This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jcb.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).


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Related Article

Heterochromatin protein 1 is recruited to various types of DNA damage
Martijn S. Luijsterburg, Christoffel Dinant, Hannes Lans, Jan Stap, Elzbieta Wiernasz, Saskia Lagerwerf, Daniël O. Warmerdam, Michael Lindh, Maartje C. Brink, Jurek W. Dobrucki, Jacob A. Aten, Maria I. Fousteri, Gert Jansen, Nico P. Dantuma, Wim Vermeulen, Leon H.F. Mullenders, Adriaan B. Houtsmuller, Pernette J. Verschure, and Roel van Driel
J. Cell Biol. 2009 185: 577-586. [Abstract] [Full Text] [PDF]





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