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Index of Online Supplemental Material for
J. Cell Biol. 10.1083/jcb.200701006
Grande-Garcia et al.
• Figure S1 Absence of Cav1 does not delay cell spreading.
• Figure S2 Constitutively active Rho and dominant-negative mutants of Rac and Cdc42 restore normal polarity in Cav1-/- fibroblasts.
• Figure S3 A dominant-negative p190RhoGAP construct rescues polarity in Cav1-/- fibroblasts.
• Figure S4 Src inhibition and p190RhoGAP knockdown in Cav1-/- MEFs restore the WT morphology pattern.
• Figure S5 Total and tyrosine phosphorylated p190RhoGAP are elevated in Cav1-/- MEFs.
• Video 1 Adhesion turnover in WT MEFs.
• Video 2 Adhesion turnover in Cav1-/- MEFS.
• Video 3 Protrusion-retraction analysis of WT MEFs.
• Video 4 Protrusion-retraction analysis of Cav1-/- MEFs.
• Video 5 Random migration of WT MEFs.
• Video 6 Random migration of Cav1-/- MEFs.
• Video 7 Random migration of Cav1-/- MEFs reconstituted with Cav1.
• Video 8 In vitro wound-healing assays in WT MEFs.
• Video 9 In vitro wound-healing assay in Cav1-/- MEFs.
• Video 10 In vitro wound-healing assay in Cav1-/- MEFs reconstituted with Cav1.
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