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Index of Online Supplemental Material for
J. Cell Biol. 10.1083/jcb.200705044
Lacalle et al.

• Figure S1 Analysis of PIPKIβ localization.

• Figure S2 Analysis of PI(4,5)P₂ and PIP₃ levels in PIPKIβ-expressing cells.

• Figure S3 Analysis of RhoA activity.

• Vidoe 1 PIPKIα does not polarize during dHL60 cell chemotaxis.

• Vidoe 2 PIPKIα does not polarize during dHL60 cell chemotaxis.

• Vidoe 3 PIPKIγ⁶³⁵ does not polarize during dHL60 cell chemotaxis.

• Vidoe 4 dHL60 cells expressing the kinase-dead PIPKIβ^K138A mutant exhibit a modest defect in uropod retraction.

• Vidoe 5 Cellular localization of the C-terminal–deleted PIPKIγ^1–502 mutant in chemotaxing dHL60 cells.

• Vidoe 6 The PIPKIβ C terminus acts as a uropod targeting sequence.

• Vidoe 7 PIPKIβwt-expressing dHL60 cells persistently polarize PI3K signaling during chemotaxis.

• Vidoe 8 Overexpression of PIPKIβΔ456 impedes polarization of PI3K signaling in directionally stimulated dHL60 cells.

• Vidoe 9 PIPKIβwt-expressing dHL60 cells persistently polarize Rac signaling during chemotaxis.

• Vidoe 10 PIPKIβΔ456 overexpression blocks polarization of Rac signaling in directionally stimulated dHL60 cells.

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